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User-recommended papers allows you to suggest a paper you would like to see on Nature India. When these papers appear on our site, users may comment and vote on them. To recommend a paper please use this form. Please note that you cannot recommend a paper you have authored. The editors will reject any self-recommendations. All comments and recommendation are checked by the editors and may be edited prior to publication on the site.

• Cell & Molecular Biology

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  • Anomalous venous blood flow and iron deposition in multiple sclerosis

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      Original article citation: J Cereb Blood Flow Metab doi:10.1038/jcbfm.2009.180 (2009)

    • Categories: Clinical Medicine, Cell & Molecular Biology, Genetics, and Neuroscience
    • Recommended by : Dr Uttara Bayani on 10/15/2009 05:33PM GMT
      
      First paper highlighting the physiological causes of neurodegeneration in Multiple Sclerosis MS. Authors have mentioned that disturbed microcirculation in jugular vein (due to pressure changes), leads to CCSVI, subsequent iron depositions in cerebrospinal region and lost neuronal motor function. The authors had actually shown in patients that corrective surgery has tremendous capacity to restores motor function in almost paralyzed MS patients.
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  • A genetically encoded photoactivatable Rac controls the motility of living cells.

    • Wu YI Hahn KM
      Original article citation: Nature 461," 104 - 108, (2008).

    • Categories: Cell & Molecular Biology and Biotechnology
    • Recommended by : KC V on 10/06/2009 10:27PM GMT
      
      Great use of biosensor based tools - elucidating whole cell influences of even subcellular activation of molecular cascades!
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  • Polo kinase and separase regulate the mitotic licensing of centriole duplication in human cells.

    • Bryan Tsou
      Original article citation: Dev cell Volume 17," 344-354 - 354, (2009).

    • Categories: Cell & Molecular Biology
    • Recommended by : Rajesh Soni on 09/25/2009 10:32PM GMT
      
      It is a very good paper to understand the centrosome duplication mechanism.
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  • Using FlyAtlas to identify better Drosophila melanogaster models of human disease

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      Original article citation: Nat Genet 39," 715 - 720, (2007).

    • Categories: Genetics, Cell & Molecular Biology, Developmental Biology, and Neuroscience
    • Recommended by : Venkat Chintapalli on 09/14/2009 08:19PM GMT
      
      This paper describes how insight into tissue specific transcriptomes could give rise to studying useful models of human disease.
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  • • 6+ votes

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  • Chromium interactions in plants: current status and future strategies

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      Original article citation: Metallomics doi:10.1039/b904571f

    • Categories: Biotechnology, Cell & Molecular Biology, and Earth & Environment
    • Recommended by : Arun K.Shanker on 08/22/2009 07:22AM GMT
      
      This mini review focuses on the recent developments in the field of Cr–plant interactions and proposes a model using a systems biology and integrated -omics approach to decipher the intricacies of Cr–plant interaction.
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  • Prediction of viral microRNA precursors based on human microRNA precursor sequence and structural features

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      Original article citation: Virol J 6," 129 , (2009).

    • Categories: Cell & Molecular Biology, Biotechnology, and Genetics
    • Recommended by : Klein K on 08/21/2009 04:22AM GMT
      
      This is an interesting paper on prediction of microRNAs from viruses using de-novo approach based on sequence and structure features.
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  • • 4+ votes

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  • Spinal Cord Repair: Bridging the Divide

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      Original article citation: Neurorehabilitation and Neural Repair 22," 429 - 437,

    • Categories: Neuroscience, Cell & Molecular Biology, and Clinical Medicine
    • Recommended by : NAGESH SHANBHAG on 05/05/2009 04:46PM GMT
      
      Unless the intrinsic and extrinsic mechanisms which are involved in the axonal regeneration post-SCI are critically deciphered, it would not lead to a substantial long-term functional recovery in the patients,despite supportive and rehabilitative strategies.
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  • • 5+ votes

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  • Interaction of APC/C-E3 ligase with Swi6/HP1 and Clr4/Suv39 in heterochromatin assembly in fission yeast

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      Original article citation: Journal of Biological Chemistry doi:10.1074/jbc.M806461200 (2008)

    • Categories: Genetics and Cell & Molecular Biology
    • Recommended by : Sneh Lata on 01/31/2009 05:53AM GMT
      
      This paper for the first time demonstrates a surprising role of Anaphase promoting Complex, APC/C in assembly of heterochromatin and silencing at mating type, centromere and rDNA loci in fission yeat.
    • Comment on this subject: 1 comment made

  • • 1+ votes

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  • Japanese Encephalitis virus infects neural progenitor cells and decreases their proliferation

    • Sulagna Das, Anirban Basu
      Original article citation: J Neurochem doi:10.1111/j.1471-4159.2008.05511.x (2008)

    • Categories: Cell & Molecular Biology and Neuroscience
    • Recommended by : hemant goldar on 06/12/2008 01:27PM GMT
      
      Japanese encephalitis virus (JEV), a common cause of encephalitis in humans, especially in children, leads to substantial neuronal injury. The survivors of JEV infection have severe cognitive impairment, motor and behavioral disorders. This paper hypothesizes that depletion of neural progenitor cells (NPCs) by the virus culminates in neurological sequelae in survivors of Japanese encephalitis (JE). It utilizes both in vivo model of JEV infection and in vitro neurosphere cultures to study progressive JEV infection. Cellular infection and cell death was determined by Flow cytometry. BrdU administration in animals and in neurospheres was used to determine the proliferative ability of NPCs. JEV leads to massive loss of actively proliferating NPC population from the subventricular zone (SVZ). The ability of JEV infected subventricular zone cells to form neurospheres is severely compromised. This can be attributed to JEV infection in NPCs, which however do not result in robust death of the resilient NPC cells. Instead, JEV suppresses the cycling ability of these cells, preventing their proliferation. JEV primarily targets at a critical post natal age and severely diminishes the NPC pool in SVZ, thus impairing the process of recovery after the insult. This arrested growth and proliferation of NPCs might have an effect on the neurological consequences in JE survivors.
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  • Sphingomyelin Functions as a Novel Receptor for Helicobacter pylori VacA

    • Vijay R. Gupta, Hetal K. Patel, Sean S. Kostolansky, Roberto A. Ballivian, Joseph Eichberg, Steven R. Blanke, Partho Ghosh
      Original article citation: PLoS Pathogens 4," e1000073 ,

    • Categories: Cell & Molecular Biology and Clinical Medicine
    • Recommended by : Anand Giddabasappa CS on 05/25/2008 07:05AM GMT
      
      Helicobacter pylori is a gastric pathogen that causes gastric and duodenal ulcer and even gastric carcinoma. VacA, a vaculating cytotoxin secreted by this pathogen is responsible for this toxic action on the host cell. Though VacA was known to interact with the cell membrane of the host (gastric epithelium), the internalization mechanisms were unknown. This paper shows that VacA binds to an unusual receptor called sphingomyelin. Sphingomyelin, a membrane lipid acts as the receptor on the host cell and facilitates binding and internalization of the cytotoxin. This article provides the molecular basis for the binding and action of VacA. Further, it also sheds light on the role of lipid rafts as target to block the pathogenic effects of H. pylori.
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